What Are the Implications of Hypoxic-Ischemic Encephalopathy?
Research indicates that hypoxic-ischemic encephalopathy is one of the leading causes of death or severe impairment among infants. HIE is also directly linked to cerebral palsy, but remains one of the rarer causes of the condition, accounting for roughly 9% of cerebral palsy cases.
The conditions and future implications of HIE will vary based on how long the child was deprived of oxygen and whether the infant experienced mild, moderate, or severe symptoms. Most of the time, newborns with mild HIE symptoms can enjoy their lives unaffected by HIE while children with severe symptoms may have a shortened lifespan as well as experience a variety of painful, debilitating complications and conditions throughout their lives.
Early symptoms of HIE include the following:
- Low heart rate
- Poor muscle tone
- Weak breathing
- Meconium-stained amniotic fluid
- Pale or bluish skin color
- Excessive acid in the blood
- Organ dysfunction, especially of the heart, lungs, liver, and kidneys
- Seizures, especially within the first 24 hours of life
When early symptoms present themselves, a wide range of tests is usually issued to determine whether or not the symptoms are due to HIE. Afterwards, a treatment course is planned which can include breathing therapy, anesthesia, oxygen treatments, and medications to control the baby’s heart functions, blood glucose levels, and seizures.
Children diagnosed with moderate to severe HIE usually need to be placed on medication for the rest of their lives and may eventually develop cognitive impairments, motor skill development delays, neurodevelopment delays, and even epilepsy or cerebral palsy.